EVOLUTIONIST CLAIMS &THE FACTS 

In previous chapters, we examined the invalidity of the theory of evolution in terms of the bodies of evidence found in fossils and from the standpoint of molecular biology. In this chapter, we will address a number of biological phenomena and concepts presented as theoretical evidence by evolutionists. These topics are particularly important for they show that there is no scientific finding that supports evolution and instead reveal the extent of the distortion and hoodwink employed by evolutionists.


Variations and Species

Variation, a term used in genetics, refers to a genetic event that causes the individuals or groups of a certain type or species to possess different characteristics from one another. For example, all the people on earth carry basically the same genetic information, yet some have slanted eyes, some have red hair, some have long noses, and others are short of stature, all depending on the extent of the variation potential of this genetic information.


DID WHALES EVOLVE FROM BEARS?
In The Origin of Species, Darwin asserted that whales had evolved from bears that tried to swim! Darwin mistakenly supposed that the possibilities of variation within a species were unlimited. 20th century science has shown this evolutionary scenario to be imaginary.

Evolutionists predicate the variations within a species as evidence to the theory. However, variation does not constitute evidence for evolution because variations are but the outcomes of different combinations of already existing genetic information and they do not add any new characteristic to the genetic information. The important thing for the theory of evolution, however, is the question of how brand-new information to make a brand-new species could come about.

Variation always takes place within the limits of genetic information. In the science of genetics, this limit is called the "gene pool". All of the characteristics present in the gene pool of a species may come to light in various ways due to variation. For example, as a result of variation, varieties that have relatively longer tails or shorter legs may appear in a certain species of reptile, since information for both long-legged and short-legged forms may exist in the gene pool that species. However, variations do not transform reptiles into birds by adding wings or feathers to them, or by changing their metabolism. Such a change requires an increase in the genetic information of the living thing, which is certainly not possible through variations.

Darwin was not aware of this fact when he formulated his theory. He thought that there was no limit to variations. In an article he wrote in 1844 he stated: "That a limit to variation does exist in nature is assumed by most authors, though I am unable to discover a single fact on which this belief is grounded".151 In The Origin of Species he cited different examples of variations as the most important evidence for his theory.

For instance, according to Darwin, animal breeders who mated different varieties of cattle in order to bring about new varieties that produced more milk, were ultimately going to transform them into a different species. Darwin's notion of "unlimited variation" is best seen in the following sentence from The Origin of Species:

I can see no difficulty in a race of bears being rendered, by natural selection, more and more aquatic in their habits, with larger and larger mouths, till a creature was produced as monstrous as a whale.152

The reason Darwin cited such a far-fetched example was the primitive understanding of science in his day. Since then, in the 20th century, science has posited the principle of "genetic stability" (genetic homeostasis), based on the results of experiments conducted on living things. This principle holds that, since all mating attempts carried out to produce new variations have been inconclusive, there are strict barriers among different species of living things. This meant that it was absolutely impossible for animal breeders to convert cattle into a different species by mating different variations of them, as Darwin had postulated.

Norman Macbeth, who disproved Darwinism in his book Darwin Retried, states:

The heart of the problem is whether living things do indeed vary to an unlimited extent... The species look stable. We have all heard of disappointed breeders who carried their work to a certain point only to see the animals or plants revert to where they had started. Despite strenuous efforts for two or three centuries, it has never been possible to produce a blue rose or a black tulip.153

Luther Burbank, considered the most competent breeder of all time, expressed this fact when he said, "there are limits to the development possible, and these limits follow a law." 154 The Danish scientist W. L. Johannsen sums the matter up this way:

The variations upon which Darwin and Wallace had placed their emphasis cannot be selectively pushed beyond a certain point, that such a variability does not contain the secret of 'indefinite departure.155

In the same way, the different finches that Darwin saw on the Galapagos Islands are another example of variation that is no evidence for "evolution". Recent observations have revealed that the finches did not undergo an unlimited variation as Darwin's theory presupposed. Moreover, most of the different types of finches which Darwin thought represented 14 distinct species actually mated with one another, which means that they were variations that belonged to the same species. Scientific observation shows that the finch beaks, which have been mythicized in almost all evolutionist sources, are in fact an example of "variation"; therefore, they do not constitute evidence for the theory of evolution. For example, Peter and Rosemary Grant, who spent years observing the finch varieties in the Galapagos Islands looking for evidence for Darwinistic evolution, were forced to conclude that no "evolution" that leads to the emergence of new traits ever takes place there.


Antibiotic Resistance and DDT Immunity are not Evidence for Evolution

One of the biological concepts that evolutionists try to present as evidence for their theory is the resistance of bacteria to antibiotics. Many evolutionist sources show antibiotic resistance as "an example of the development of living things by advantageous mutations". A similar claim is also made for the insects which build immunity to insecticides such as DDT.

However, evolutionists are mistaken on this subject too.

Antibiotics are "killer molecules" that are produced by micro-organisms to fight other micro-organisms. The first antibiotic was penicillin, discovered by Alexander Fleming in 1928. Fleming realized that mould produced a molecule that killed the Staphylococcus bacterium, and this discovery marked a turning point in the world of medicine. Antibiotics derived from micro-organisms were used against bacteria and the results were successful.

Soon, something new was discovered. Bacteria build immunity to antibiotics over time. The mechanism works like this: A large proportion of the bacteria that are subjected to antibiotics die, but some others, which are not affected by that antibiotic, replicate rapidly and soon make up the whole population. Thus, the entire population becomes immune to antibiotics.

Evolutionists try to present this as "the evolution of bacteria by adapting to conditions".

The truth, however, is very different from this superficial interpretation. One of the scientists who has done the most detailed research into this subject is the Israeli biophysicist Lee Spetner, who is also known for his book Not by Chance published in 1997. Spetner maintains that the immunity of bacteria comes about by two different mechanisms, but neither of them constitutes evidence for the theory of evolution. These two mechanisms are:

1) The transfer of resistance genes already extant in bacteria.

2) The building of resistance as a result of losing genetic data because of mutation.

Professor Spetner explains the first mechanism in an article published in 2001:

Some microorganisms are endowed with genes that grant resistance to these antibiotics. This resistance can take the form of degrading the antibiotic molecule or of ejecting it from the cell... The organisms having these genes can transfer them to other bacteria making them resistant as well. Although the resistance mechanisms are specific to a particular antibiotic, most pathogenic bacteria have... succeeded in accumulating several sets of genes granting them resistance to a variety of antibiotics.156

Spetner then goes on to say that this is not "evidence for evolution":

The acquisition of antibiotic resistance in this manner... is not the kind that can serve as a prototype for the mutations needed to account for Evolution. The genetic changes that could illustrate the theory must not only add information to the bacterium's genome, they must add new information to the biocosm. The horizontal transfer of genes only spreads around genes that are already in some species.157

So, we cannot talk of any evolution here, because no new genetic information is produced: genetic information that already exists is simply transferred between bacteria.

The second type of immunity, which comes about as a result of mutation, is not an example of evolution either. Spetner writes:

...A microorganism can sometimes acquire resistance to an antibiotic through a random substitution of a single nucleotide... Streptomycin, which was discovered by Selman Waksman and Albert Schatz and first reported in 1944, is an antibiotic against which bacteria can acquire resistance in this way. But although the mutation they undergo in the process is beneficial to the microorganism in the presence of streptomycin, it cannot serve as a prototype for the kind of mutations needed by NDT[Neo Darwinian Theory]. The type of mutation that grants resistance to streptomycin is manifest in the ribosome and degrades its molecular match with the antibiotic molecule. This change in the surface of the microorganism's ribosome prevents the streptomycin molecule from attaching and carrying out its antibiotic function. It turns out that this degradation is a loss of specificity and therefore a loss of information. The main point is that (Evolution) cannot be achieved by mutations of this sort, no matter how many of them there are. Evolution cannot be built by accumulating mutations that only degrade specificity.158

To sum up, a mutation impinging on a bacterium's ribosome makes that bacterium resistant to streptomycin. The reason for this is the "decomposition" of the ribosome by mutation. That is, no new genetic information is added to the bacterium. On the contrary, the structure of the ribosome is decomposed, that is to say, the bacterium becomes "disabled". (Also, it has been discovered that the ribosome of the mutated bacterium is less functional than that of normal bacterium). Since this "disability" prevents the antibiotic from attaching onto the ribosome, "antibiotic resistance" develops.

Finally, there is no example of mutation that "develops the genetic information".

The same situation holds true for the immunity that insects develop to DDT and similar insecticides. In most of these instances, immunity genes that already exist are used. The evolutionist biologist Francisco Ayala admits this fact, saying, "The genetic variants required for resistance to the most diverse kinds of pesticides were apparently present in every one of the populations exposed to these man-made compounds."159 Some other examples explained by mutation, just as with the ribosome mutation mentioned above, are phenomena that cause "genetic information deficit" in insects.

In this case, it cannot be claimed that the immunity mechanisms in bacteria and insects constitute evidence for the theory of evolution. That is because the theory of evolution is based on the assertion that living things develop through mutations. However, Spetner explains that neither antibiotic immunity nor any other biological phenomena indicate such an example of mutation:

The mutations needed for macroevolution have never been observed. No random mutations that could represent the mutations required by Neo-Darwinian Theory that have been examined on the molecular level have added any information. The question I address is: Are the mutations that have been observed the kind the theory needs for support? The answer turns out to be NO!160


The Fallacy of Vestigial Organs

For a long time, the concept of "vestigial organs" appeared frequently in evolutionist literature as "evidence" of evolution. Eventually, it was silently put to rest when this was proved to be invalid. But some evolutionists still believe in it, and from time to time someone will try to advance "vestigial organs" as important evidence of evolution.

The notion of "vestigial organs" was first put forward a century ago. As evolutionists would have it, there existed in the bodies of some creatures a number of non-functional organs. These had been inherited from progenitors and had gradually become vestigial from lack of use.


All instances of vestigial organs have been disproved in time. For example the semicircular fold in the eye, which was mentioned in the Origins as a vestigial structure, has been shown to be fully functional in our time, though its function was unknown in Darwin's time. This organ lubricates the eyeball.

The whole assumption is quite unscientific, and is based entirely on insufficient knowledge. These "non-functional organs" were in fact organs whose "functions had not yet been discovered". The best indication of this was the gradual yet substantial decrease in evolutionists' long list of vestigial organs. S.R. Scadding, an evolutionist himself, concurred with this fact in his article "Can vestigial organs constitute evidence for evolution?" published in the journal Evolutionary Theory:

Since it is not possible to unambiguously identify useless structures, and since the structure of the argument used is not scientifically valid, I conclude that "vestigial organs" provide no special evidence for the theory of evolution.161

The list of vestigial organs that was made by the German Anatomist R. Wiedersheim in 1895 included approximately 100 organs, including the appendix and coccyx. As science progressed, it was discovered that all of the organs in Wiedersheim's list in fact had very important functions. For instance, it was discovered that the appendix, which was supposed to be a "vestigial organ", was in fact a lymphoid organ that fought against infections in the body. This fact was made clear in 1997: "Other bodily organs and tissues-the thymus, liver, spleen, appendix, bone marrow, and small collections of lymphatic tissue such as the tonsils in the throat and Peyer's patch in the small intestine-are also part of the lymphatic system. They too help the body fight infection."162

It was also discovered that the tonsils, which were included in the same list of vestigial organs, had a significant role in protecting the throat against infections, particularly until adolescence. It was found that the coccyx at the lower end of the vertebral column supports the bones around the pelvis and is the convergence point of some small muscles and for this reason, it would not be possible to sit comfortably without a coccyx. In the years that followed, it was realised that the thymus triggered the immune system in the human body by activating the T cells, that the pineal gland was in charge of the secretion of some important hormones, that the thyroid gland was effective in providing steady growth in babies and children, and that the pituitary gland controlled the correct functioning of many hormone glands. All of these were once conside-red to be "vestigial organs". Finally, the semi-lunar fold in the eye, which was referred to as a vestigial organ by Darwin, has been found in fact to be in charge of cleansing and lubricating the eyeball.

There was a very important logical error in the evolutionist claim regarding vestigial organs. As we have just seen, this claim was that the vestigial organs in living things were inherited from their ancestors. However, some of the alleged "vestigial" organs are not found in the species alleged to be the ancestors of human beings! For example, the appendix does not exist in some ape species that are said to be ancestors of man. The famous biologist H. Enoch, who challenged the theory of vestigial organs, expressed this logical error as follows:

Apes possess an appendix, whereas their less immediate relatives, the lower apes, do not; but it appears again among the still lower mammals such as the opossum. How can the evolutionists account for this?163

Simply put, the scenario of vestigial organs put forward by evolutionists contains a number of serious logical flaws, and has in any case been proven to be scientifically untrue. There exists not one inherited vestigial organ in the human body, since human beings did not evolve from other creatures as a result of chance, but were created in their current, complete, and perfect form.

151 Loren C. Eiseley, The Immense Journey, Vintage Books, 1958, p. 186.
152 Charles Darwin, The Origin of Species: A Facsimile of the First Edition, Harvard University Press, 1964, p. 184.
153 Norman Macbeth, Darwin Retried: An Appeal to Reason, Harvard Common Press, New York: 1971, p. 33.
154 Ibid, p. 36.
155 Loren Eiseley, The Immense Journey, Vintage Books, 1958. p. 227.
156 Dr. Lee Spetner, "Lee Spetner/Edward Max Dialogue: Continuing an exchange with Dr. Edward E. Max", 2001, http://www.trueorigin.org/spetner2.asp.
157 Ibid.
158 Ibid.
159 Francisco J. Ayala, "The Mechanisms of Evolution", Scientific American, Vol 239, September 1978, p. 64.
160 Dr. Lee Spetner, "Lee Spetner/Edward Max Dialogue: Continuing an exchange with Dr. Edward E. Max", 2001, http://www.trueorigin.org/spetner2.asp.
161 S. R. Scadding, "Do 'Vestigial Organs' Provide Evidence for Evolution?", Evolutionary Theory, Vol 5, May 1981, p. 173.
162 The Merck Manual of Medical Information, Home edition, New Jersey: Merck & Co., Inc. The Merck Publishing Group, Rahway, 1997
163 H. Enoch, Creation and Evolution, New York: 1966, p. 18-19.